SAMD00324913
External Id
NODAI Genome Research Center, Tokyo University of Agriculture
INSDC center name
2023-04-26T00:00:00Z
INSDC first public
2024-06-02T07:55:50.883Z
INSDC last update
DRS302148
INSDC secondary accession
live
INSDC status
MIGS.eu
NCBI submission model
MIGS.eu.6.0
NCBI submission package
DRS302148
SRA accession
2016-04-07
collection_date
Plants often encounter osmotic stress caused by drought, cold, or salinity and have evolved acquired tolerance systems against more severe stresses. We previously found that acquired osmotolerance induced after salt stress is widespread among Arabidopsis thaliana accessions and identified ACQOS as the responsible gene, causing detrimental autoimmunity via ENHANCED DISEASE SUSCEPTIBILITY1 (EDS1) and PHYTOALEXIN DEFICIENT4 (PAD4) under osmotic stress, thereby reducing osmotolerance. However, it remains unclear how the tolerance is established. To reveal the mechanism underlying the acquired osmotolerance, we here isolated an acquired osmotolerance-defective (aod) 13 mutant by screening 30,000 M2 seedlings obtained from ion beam-mutagenized seeds of Bu-5, an accession with acquired osmotolerance.
description
agar medium
env_medium
120Mb
estimated_size
Germany: Burghaun/Rhon)
geo_loc_name
Arabidopsis thaliana
http://purl.obolibrary.org/obo/NCBITaxon_3702
organism
diploid
ploidy
Genome sequence of Arabidopsis thaliana Bu-5 wild type and the mutant
project_name
sexual
propagation
Bu-5
sample_name
Genome sequence of Arabidopsis thaliana Bu-5 wild type
title