The biochemical pathology of aspirin-induced gastric damage.
D000367:Age Factors; D000428:Alcohol Drinking; D000818:Animals; D000893:Anti-Inflammatory Agents / Q000009:adverse effects; D001206:Ascorbic Acid Deficiency / Q000503:physiopathology; D001241:Aspirin / Q000008:administration & dosage / Q000009:adverse effects*; D001789:Blood Group Antigens; D005260:Female; D005502:Food; D005753:Gastric Mucosa / Q000098:blood supply; D006471:Gastrointestinal Hemorrhage / Q000503:physiopathology; D006801:Humans; D006863:Hydrogen-Ion Concentration; D008297:Male; D009747:Nutritional Physiological Phenomena; D010316:Particle Size; D010539:Permeability; D011453:Prostaglandins / Q000502:physiology; D012039:Regional Blood Flow; D012459:Salicylates / Q000009:adverse effects; D012737:Sex Factors; D013272:Stomach Diseases / Q000139:chemically induced* / Q000503:physiopathology; D013276:Stomach Ulcer / Q000139:chemically induced; D013329:Structure-Activity Relationship
D000893:Anti-Inflammatory Agents; D001789:Blood Group Antigens; D011453:Prostaglandins; D012459:Salicylates; D001241:Aspirin