[Physiopathology of low cardiac outputs after ECC and their treatment].
Etiologic circumtances of low cardiac outputs are studied according as they concern a diminution of the precharge (hypovolemy of different reasons), an augmentation of the postcharge (by augmentation of aortic impedance or resistance to blood flow) or a cardiac deficiency. The latter can be of non-ischemic origin (effects of beta-blocking drugs, cardiac arrhytmias tamponade, deficience of myocardiac fiber, metabolic disease) or ischemic:--during E.C.C.: diminution of available oxygen (hypothermial), arterial pressure and output too low, clamping time or ventricular fibrillation too long;--after E.C.C.: insufficient surgical correction, coronary embolism lack of balance between apport and expense of oxygen. Therapeutics ways are discussed regarding the different etiologies. The central rule is that it is better to relieve an ischemic myocardia than lash it. But treatment of low cardiac outputs is most often an average solution which must be choose in regard to a well understood physiopathology.
D000319:Adrenergic beta-Antagonists / Q000009:adverse effects / Q000627:therapeutic use; D001794:Blood Pressure; D001810:Blood Volume; D002302:Cardiac Output; D006348:Cardiac Surgical Procedures / Q000009:adverse effects; D003331:Coronary Vessels; D005112:Extracorporeal Circulation / Q000009:adverse effects; D006801:Humans; D007511:Ischemia / Q000150:complications / Q000628:therapy; D014693:Ventricular Fibrillation / Q000150:complications
D000319:Adrenergic beta-Antagonists
Physiopathologie des bas débits cardiaques post cec et leur traitement