Inhalation toxicology of automotive emissions as affected by an oxidation exhaust catalyst.
Preliminary data are given on the acute inhalation toxicology of automotive emissions as affected by an oxidation exhaust catalyst. The catalyst effectively reduced CO and HC in the exhause which apparently had an effect (at least in a closed exposure system) on oxidant and NO2 levels by altering the HC/NOx ratio. There was a resultant reduction in biological effects due to the exposure. The catalyst altered the type of particulate to one which probably contained sulfuric acid as a major component. No evidence was present in these acute exposures to suggest a toxic response due to the higher sulfate emissions or possible catalyst attrition products. The effects of long-term exposure have not yet been investigated.
D000397:Air Pollution / Q000517:prevention & control; D000818:Animals; D001336:Automobiles / Q000592:standards; D002248:Carbon Monoxide / Q000633:toxicity*; D002384:Catalysis; D004782:Environmental Health; D004864:Equipment and Supplies; D004912:Erythrocytes / Q000187:drug effects; D005260:Female; D006461:Hemolysis / Q000187:drug effects; D006838:Hydrocarbons / Q000633:toxicity*; D007668:Kidney / Q000473:pathology; D008099:Liver / Q000473:pathology; D008168:Lung / Q000473:pathology; D008297:Male; D010165:Palladium / Q000378:metabolism; D010984:Platinum / Q000378:metabolism; D051381:Rats; D001335:Vehicle Emissions / Q000633:toxicity*
D006838:Hydrocarbons; D001335:Vehicle Emissions; D010984:Platinum; D010165:Palladium; D002248:Carbon Monoxide