Effect of acetate upon arterial gases.
Following infusion of sodium acetate, a significant fall in arterial oxygen tension (PO2) was observed (p less than 0.001). A rise in PCO2 or bicarbonate was also statistically significant. pH was however remained unchanged. Though a rise in PCO2, bicarbonate, pH was observed following infusion of sodium bicarbonate, no significant fall in PO2 was observed. These results suggest a role of acetate ion for the fall of arterial oxygen tension; the hemodialysis-induced hypoxemia.
D000085:Acetates / Q000008:administration & dosage*; D001249:Asthma / Q000209:etiology; D001639:Bicarbonates / Q000008:administration & dosage; D002245:Carbon Dioxide / Q000097:blood*; D005260:Female; D006801:Humans; D006863:Hydrogen-Ion Concentration; D000860:Hypoxia / Q000209:etiology; D008297:Male; D010100:Oxygen / Q000097:blood*; D006435:Renal Dialysis
D000085:Acetates; D001639:Bicarbonates; D002245:Carbon Dioxide; D010100:Oxygen