Gentamicin resistance in Staphylococcus aureus.
A clinical isolate of Staphylococcus aureus was resistant to gentamicin, kanamycin, and tobramycin by virtue of the production of a drug-inactivating enzyme. Attempts to establish the cellular location of the genetic determinants for enzyme synthesis have given equivocal results.
D000900:Anti-Bacterial Agents / Q000494:pharmacology*; D004352:Drug Resistance, Microbial; D005839:Gentamicins / Q000494:pharmacology*; D006801:Humans; D007612:Kanamycin / Q000494:pharmacology; D009355:Neomycin / Q000494:pharmacology; D013210:Staphylococcus / Q000201:enzymology; D013211:Staphylococcus aureus / Q000187:drug effects*; D013307:Streptomycin / Q000494:pharmacology; D014031:Tobramycin / Q000494:pharmacology
D000900:Anti-Bacterial Agents; D005839:Gentamicins; D007612:Kanamycin; D009355:Neomycin; D014031:Tobramycin; D013307:Streptomycin