Role of Staphylococcus aureus cell wall antigens in the stimulation of delayed hypersensitivity after staphylococcal infection.
In Staphylococcus aureus-infected mice, prior treatment with cyclophosphamide enhanced delayed hypersensitivity to cell walls and peptidoglycan. S. aureus did not cross react with S. epidermidis or S. saprophyticus.
D000818:Animals; D000942:Antigens, Bacterial; D002473:Cell Wall / Q000276:immunology; D003429:Cross Reactions; D000939:Epitopes; D006968:Hypersensitivity, Delayed; D051379:Mice; D010457:Peptidoglycan / Q000276:immunology; D013203:Staphylococcal Infections / Q000276:immunology*; D013205:Staphylococcal Protein A / Q000276:immunology; D013211:Staphylococcus aureus / Q000276:immunology*; D013682:Teichoic Acids / Q000276:immunology
D000942:Antigens, Bacterial; D000939:Epitopes; D010457:Peptidoglycan; D013205:Staphylococcal Protein A; D013682:Teichoic Acids