[Pathophysiology of delirium].
Based on a series of known facts on clinical findings and changes in the metabolism of chronic alcoholics and delirious people the possible pathomechanism of cerebral imbalances is presented according to a synopsis. The clinical symptomatology, in particular vegetative symptoms and the coordination of reflexes - to which more attention should be paid than has been up to now - make delirium appear a diencephalic illness, the sympathicotonic and ergotropic development of which together with a preponderance of noradrenergic-adrenergic mechanisms, remains unexplained to this day.
D000302:Adrenal Cortex / Q000503:physiopathology; D000430:Alcohol Withdrawal Delirium / Q000503:physiopathology*; D000437:Alcoholism / Q000503:physiopathology; D001786:Blood Glucose / Q000378:metabolism; D001921:Brain / Q000503:physiopathology; D004837:Epinephrine / Q000097:blood; D005943:Gluconeogenesis; D006019:Glycolysis; D006801:Humans; D008099:Liver / Q000503:physiopathology; D009474:Neurons / Q000502:physiology; D009638:Norepinephrine / Q000097:blood; D010625:Phenylethanolamine N-Methyltransferase / Q000502:physiology; D011604:Psychoses, Alcoholic / Q000503:physiopathology*
D001786:Blood Glucose; D010625:Phenylethanolamine N-Methyltransferase; D009638:Norepinephrine; D004837:Epinephrine
Bemerkungen zur Pathophysiologie des Delirs.