Effect of ultrasonic treatment on the organs of experimental animals. III. Enzyme-histochemical examination of the prolonged effect.
The protracted effect of ultrasound treatment on the parenchymatous organs of rats has been studied histochemically at 1 to 10-day intervals. As a result of the altered redox conditions it is the oxidative enzymes in the first place that react to ultrasound treatment and their normalization takes a long time. Exposure decreased the activity of lysosomal enzymes and in the cell membrane the alkaline phosphatase and ATP-ase too. The activity of hydrolases returned to normal parallel with the normalization of the microscopic structure.
D000251:Adenosine Triphosphatases / Q000378:metabolism; D000469:Alkaline Phosphatase / Q000378:metabolism; D000626:Aminopeptidases / Q000378:metabolism; D000818:Animals; D007668:Kidney / Q000378:metabolism*; D008099:Liver / Q000378:metabolism*; D008112:Liver Glycogen / Q000378:metabolism; D008247:Lysosomes / Q000201:enzymology; D010088:Oxidoreductases / Q000378:metabolism; D051381:Rats; D013154:Spleen / Q000378:metabolism*; D014465:Ultrasonics / Q000009:adverse effects*
D008112:Liver Glycogen; D010088:Oxidoreductases; D000469:Alkaline Phosphatase; D000626:Aminopeptidases; D000251:Adenosine Triphosphatases