[Cardiac glycoside receptor affinity and (Na++K+)-ATPase inhibition of digitoxin, digoxin, methyldigoxin, strophanthin, proscillaridin, and meproscillarin. Measurements on isolated human cardiac cell membranes].
D000251:Adenosine Triphosphatases / Q000037:antagonists & inhibitors*; D002301:Cardiac Glycosides / Q000378:metabolism / Q000494:pharmacology*; D002462:Cell Membrane / Q000378:metabolism; D004074:Digitoxin / Q000378:metabolism / Q000494:pharmacology; D004077:Digoxin / Q000031:analogs & derivatives / Q000378:metabolism / Q000494:pharmacology; D006801:Humans; D066298:In Vitro Techniques; D009206:Myocardium / Q000378:metabolism*; D011442:Proscillaridin / Q000378:metabolism / Q000494:pharmacology; D011955:Receptors, Drug / Q000378:metabolism*; D013328:Strophanthins / Q000378:metabolism / Q000494:pharmacology
D002301:Cardiac Glycosides; D011955:Receptors, Drug; D013328:Strophanthins; D004077:Digoxin; D004074:Digitoxin; D000251:Adenosine Triphosphatases; D011442:Proscillaridin
Vergleichende Messungen der Herzglykosid-Rezeptoraffinität und der Hemmung der (Na+ + K+)-ATPase durch Digitoxin, Digoxin, Methyldigoxin, Strophanthin, Proscillaridin und Meproscillarin an isolierten menschlichen Herzmuskelzellmembranen.