[Vessels of the heart and lung in some experimental defects].
Under study were changes of intraorganic blood vessels of the heart and lungs in some experimental defects (open arterial defect, coarctation of the aorta, simultaneous existence of these two defects, stenosis of the pulmonary trunk, defect of the interatrial septum, triad of Fallot, syndrom of Lutembachet). Morphological data correlated with blood pressure in the pulmonary circulation and cardiac chambers. The complex of compensatory-adaptational mechanisms consisting of comparatively active and passive zones is formed in the heart and lungs. In most cases the changes develop in the vessels already existing. In hypertrophy of the myocardium when there is hypertension and hypervolemia in coronary vessels, sinusoids perform the function of blood reservoir, to a certain degree balancing the blood pressure, and luminar ducts relieve the muscle from excessive blood. The changes in the vascular system of the lung are directly dependent upon the pressure in the pulmonary circulation and the duration of observation. The closing arteries are the most active link in the chain of compensatory-adaptational mechanisms.
D000818:Animals; D001017:Aortic Coarctation / Q000473:pathology; D001794:Blood Pressure; D006332:Cardiomegaly / Q000473:pathology; D003331:Coronary Vessels / Q000473:pathology*; D004285:Dogs; D006330:Heart Defects, Congenital / Q000473:pathology*; D006344:Heart Septal Defects, Atrial / Q000473:pathology; D008168:Lung / Q000098:blood supply*; D008185:Lutembacher Syndrome / Q000473:pathology; D011651:Pulmonary Artery / Q000473:pathology; D011666:Pulmonary Valve Stenosis / Q000473:pathology*; D013771:Tetralogy of Fallot / Q000473:pathology
Sosudy serdtsa i legkogo pri nekotorykh éksperimental'nykh porokakh