[Effect of experiment hypoxia on the ATPase activity of brain, liver and myocardial nuclei and mitochondria].
Effect of circulatory, hypoxic and cytotoxic hypoxia on the ATPase activity was studied in mitochondrial fractions of brain, heart and liver tissues and in nuclear fractions of brain and liver tissues. All types of oxygen deficiency caused alterations in the activity of enyzmes studied. The observed alterations in the ATPase activity possessed the distinct phase type.
D000251:Adenosine Triphosphatases / Q000378:metabolism*; D000818:Animals; D001921:Brain / Q000201:enzymology*; D002467:Cell Nucleus / Q000201:enzymology; D004140:Dinitrophenols; D000860:Hypoxia / Q000201:enzymology*; D008099:Liver / Q000201:enzymology*; D008274:Magnesium; D008928:Mitochondria / Q000201:enzymology; D008930:Mitochondria, Liver / Q000201:enzymology; D008931:Mitochondria, Muscle / Q000201:enzymology; D009206:Myocardium / Q000201:enzymology*; D051381:Rats
D004140:Dinitrophenols; D000251:Adenosine Triphosphatases; D008274:Magnesium
Vliianie éksperimental'noĭ gipoksii na ATF-aznuiu aktivnost' iader i mitokhondriĭ mozga, pecheni i serdtsa.