Effects of severe hemodynamic pressure overload on the properties of canine left ventricular myosin: mechanism by which myosin ATPase activity is lowered during chronic increased hemodynamic stress.
D000251:Adenosine Triphosphatases / Q000378:metabolism*; D000641:Ammonia / Q000494:pharmacology; D000818:Animals; D001024:Aortic Valve Stenosis / Q000378:metabolism; D001665:Binding Sites; D001794:Blood Pressure; D002118:Calcium / Q000494:pharmacology; D004285:Dogs; D004789:Enzyme Activation / Q000187:drug effects; D006352:Heart Ventricles / Q000201:enzymology; D007700:Kinetics; D009206:Myocardium / Q000378:metabolism; D009218:Myosins / Q000378:metabolism*; D011188:Potassium / Q000494:pharmacology; D016276:Ventricular Function
D000641:Ammonia; D000251:Adenosine Triphosphatases; D009218:Myosins; D011188:Potassium; D002118:Calcium