Structural alteration and destructive corneal disease.
There is good evidence that maintenance of the structural integrity of the human cornea carries with it significant advantages over and above the preservation of normal function. The breakdown in the protective layers of the cornea and invasion by blood vessels and lymphatics would seem to add greatly to the risk of recurrent inflammatory disease. There is a solid rationale, therefore, for the development of therapeutic principles aimed at preventing these structural changes from occurring.
D000293:Adolescent; D000328:Adult; D000818:Animals; D000936:Antigen-Antibody Complex; D003315:Cornea / Q000098:blood supply / Q000276:immunology / Q000473:pathology; D003316:Corneal Diseases / Q000150:complications* / Q000473:pathology; D004848:Epithelium / Q000473:pathology; D005260:Female; D006801:Humans; D007105:Immune Complex Diseases / Q000150:complications; D007634:Keratitis / Q000209:etiology*; D008297:Male; D008875:Middle Aged; D011817:Rabbits; D013666:Tears
D000936:Antigen-Antibody Complex