[Increased transmission at electronic synapses and paroxysmal depolarization induced by cardiazol in the Mauthner cell].
Cardiazol induces in the Mauthner cell the paroxysmal depolarizing shifts which are characteristic of epilepsia at the neuronal level. The period of depolarizations is preceded and, later, accompanied by an increased transmission at the electrotonic synapses which are established upon this neuron by primary afferent vestibular fibers. Increased excitability of the chemical synapses occurs subsequently.
D000818:Animals; D006054:Goldfish; D008564:Membrane Potentials / Q000187:drug effects*; D009474:Neurons / Q000187:drug effects; D010433:Pentylenetetrazole / Q000494:pharmacology*; D012640:Seizures / Q000503:physiopathology; D009435:Synaptic Transmission / Q000187:drug effects*; D013687:Telencephalon
D010433:Pentylenetetrazole
Transmission accrue aux synapses électrotoniques et dépolarisations paroxystiques produites par le cardiazol au niveau de la cellule de Mauthner