Somatosensory cerebral evoked potentials after vascular lesions of the brain-stem and diencephalon.
Clinical and electrophysiological observations are described in 7 patients with clinically well-identified vascular lesions of the brain-stem or diencephalon. In the patients of Group A with a thalamic syndrome, the somatosensory cerebral evoked potentials had a reduced voltage and increased latency on the affected side. No significant anomalies were recorded in the patients of Group B with a Wallenberg or Weber syndrome. In patients of group C with a locked-in syndrome, the cerebral evoked potentials presented marked bilateral anomalies which provided interesting data about the extension of the pontine vascular lesions into the tegmentum. The pathophysiological mechanisms involved in the changes of average cerebral evoked potentials and in the slowing of corticipetal conduction are discussed.
D000328:Adult; D000368:Aged; D001933:Brain Stem / Q000098:blood supply*; D002543:Cerebral Hemorrhage / Q000503:physiopathology; D002561:Cerebrovascular Disorders / Q000503:physiopathology*; D004027:Diencephalon / Q000098:blood supply*; D004558:Electric Stimulation; D005071:Evoked Potentials; D005260:Female; D006801:Humans; D007238:Infarction / Q000503:physiopathology; D014854:Lateral Medullary Syndrome / Q000503:physiopathology; D008297:Male; D008875:Middle Aged; D010525:Peripheral Nerves / Q000503:physiopathology; D011149:Pons / Q000098:blood supply; D011782:Quadriplegia / Q000503:physiopathology; D011930:Reaction Time; D012640:Seizures / Q000503:physiopathology; D013003:Somatosensory Cortex / Q000503:physiopathology*; D009435:Synaptic Transmission; D013577:Syndrome; D013788:Thalamus / Q000098:blood supply