Aldosterone regulation in anephric patients.
The response of plasma aldosterone to hemodialysis, 3 h orthostatism, K-loading and angiotensin II and ACTH infusions has been studied. Hemodialysis, orthostatism and angiotensin II infusion do not modify aldosterone levels. By the contrary ACTH and potassium originate a significant increase in plasma aldosterone. They seem to be the main aldosterone secretion regulators in the absence of renin production.
D000324:Adrenocorticotropic Hormone / Q000494:pharmacology; D000450:Aldosterone / Q000097:blood / Q000378:metabolism*; D000804:Angiotensin II / Q000494:pharmacology*; D006801:Humans; D007668:Kidney / Q000502:physiology* / Q000601:surgery; D009392:Nephrectomy; D011187:Posture; D011188:Potassium / Q000494:pharmacology; D006435:Renal Dialysis; D012636:Secretory Rate / Q000187:drug effects
D000804:Angiotensin II; D000450:Aldosterone; D000324:Adrenocorticotropic Hormone; D011188:Potassium