title: [Calcitonin, gastrin, parathyroid hormone and the autonomic nervous system].
abstract: Stimulation of the beta-adrenergic terminations was employed to determine whether calcitonin-secreting thyroid C cells are derived from the neural crest. Calcitonin secretion was increased, whereas parathormone, insulin and gastrin values were not significantly changed. In addition, administration of a beta-blocking drug before ethanol led to a marked fall in calcitonin with respect to the baseline. Once again, other hormone levels were not affected.
mesh_terms: D002116:Calcitonin / Q000378:metabolism*; D005755:Gastrins / Q000378:metabolism*; D006801:Humans; D010281:Parathyroid Hormone / Q000378:metabolism*; D011433:Propranolol / Q000494:pharmacology; D011941:Receptors, Adrenergic / Q000502:physiology*; D011943:Receptors, Adrenergic, beta / Q000502:physiology*; D006435:Renal Dialysis; D014511:Uremia / Q000503:physiopathology
chemical_list: D005755:Gastrins; D010281:Parathyroid Hormone; D011941:Receptors, Adrenergic; D011943:Receptors, Adrenergic, beta; D002116:Calcitonin; D011433:Propranolol
vernacular_title: Calcitonima, gastrina, paratormone, sistema nervoso autonomo.