Type C retrovirus activation and possible functions in the normal and tumor-bearing host.
The pathological consequences of tumor virus infection, transformation, and tumor development in certain experimental animals is a well-established and accepted fact. More recently, it has been suggested that these viruses may also have a physiological function participating in such processes as cellular differentiation, immune recognition, and embryogenesis. This paper delineates the current information giving some credence to physiological function for type C viruses.
D000437:Alcoholism / Q000150:complications; D000818:Animals; D045166:Estradiol Congeners / Q000494:pharmacology; D006801:Humans; D007109:Immunity; D051379:Mice; D009369:Neoplasms / Q000209:etiology; D009374:Neoplasms, Experimental / Q000382:microbiology*; D012190:Retroviridae / Q000276:immunology; D014412:Tumor Virus Infections / Q000382:microbiology*; D014764:Viral Proteins / Q000378:metabolism; D014779:Virus Replication / Q000187:drug effects
D045166:Estradiol Congeners; D014764:Viral Proteins