Mechanisms of heterotypic immunity against canine distemper.
Hep-2 cells infected with measles virus (MV) for as short as 6 h became refractory to superinfection with canine distemper virus (CDV) but not to vesicular stomatitis virus (VSV). The exact mechanism of such interference is unknown but probably occurs after virus attachment and penetration. These results verify the suggestion that virus interference may be a mechanism of heterotypic protection against canine distemper.
D000818:Animals; D000910:Antibodies, Heterophile; D002460:Cell Line; D004216:Distemper / Q000276:immunology*; D004285:Dogs; D007109:Immunity; D008114:Liver Neoplasms, Experimental / Q000276:immunology; D008459:Measles virus / Q000276:immunology; D051381:Rats; D014721:Vesicular stomatitis Indiana virus / Q000276:immunology
D000910:Antibodies, Heterophile