[Pathology of the bone pocket].
Inflammatory periodontal destruction includes periodontal bone destruction. In addition to etiologic factors which contribute to the formation of gingival pockets, cell-mediated immune reactions apparently play a special role in osteoclastic processes. Apart from coincidental events, the form of osteoclasia is influenced in the beginning by the form and position of the tooth. Vertical osteoclasia, a variant form, is more frequently observed in connection with juvenile periodontitis. Occlusal periodontal trauma and general primary diseases may possibly also influence the pattern of periodontal bone destruction.
D001427:Bacterial Toxins; D001862:Bone Resorption / Q000209:etiology; D003602:Cytotoxicity, Immunologic; D003731:Dental Caries / Q000209:etiology*; D003769:Dental Occlusion, Traumatic / Q000209:etiology; D003773:Dental Plaque / Q000276:immunology; D006801:Humans; D008222:Lymphokines; D008310:Malocclusion / Q000150:complications; D010514:Periodontal Pocket / Q000150:complications* / Q000276:immunology; D010518:Periodontitis / Q000150:complications*
D001427:Bacterial Toxins; D008222:Lymphokines
Die Pathologie der Knochentasche.