[On the antiviral effect of a triazole compound (author's transl)].
4-[Hydroxy-3-piperid-N-yl-prop-1-yl]-5-methyl-2-phenyl-triazole (HMPT) reduced the plaque formation caused by M 12, f 2, and Q beta and retarded the liberation of phages. The concentration of tobacco mosaic virus in primarily infected tobacco leaves was decreased. HMPT inhibited the development of local lesions. Free phages and TMV particles werenot influenced by HMPT.
D000327:Adsorption; D000998:Antiviral Agents; D003090:Coliphages / Q000187:drug effects* / Q000254:growth & development; D004926:Escherichia coli; D010880:Piperidines / Q000494:pharmacology; D010947:Plants, Toxic; D012317:RNA Phages / Q000187:drug effects* / Q000254:growth & development; D014026:Nicotiana / Q000382:microbiology; D014027:Tobacco Mosaic Virus / Q000187:drug effects* / Q000254:growth & development; D014230:Triazoles / Q000494:pharmacology*; D014779:Virus Replication / Q000187:drug effects
D000998:Antiviral Agents; D010880:Piperidines; D014230:Triazoles; C025677:4-(hydroxy-3-piperidylpropyl)-5-methyl-2-phenyltriazole
Zur antiviralen Wirkung einer Triazolverbindung.