Interruption of the renin-angiotensin system in hypertensive patients by captopril induces sustained reduction in aldosterone secretion, potassium retention and natruiresis.
D000450:Aldosterone / Q000097:blood / Q000378:metabolism / Q000652:urine; D000804:Angiotensin II / Q000097:blood*; D001794:Blood Pressure / Q000187:drug effects; D002216:Captopril / Q000494:pharmacology*; D006801:Humans; D006973:Hypertension / Q000097:blood*; D009318:Natriuresis; D011188:Potassium / Q000378:metabolism; D011392:Proline / Q000031:analogs & derivatives*; D012078:Renal Artery Obstruction / Q000097:blood; D012083:Renin / Q000097:blood*; D012964:Sodium / Q000378:metabolism
D000804:Angiotensin II; D000450:Aldosterone; D011392:Proline; D002216:Captopril; D012964:Sodium; D012083:Renin; D011188:Potassium