[The hyposmolar-hyponatremic syndrome in hepatic cirrhosis. Possible role of ADH].
Following some remarks on the hyposomolar-hyponatraemic syndrome and on the formation of free water, the possible aetiopathogenetic mechanisms of hyponatraemia in ascitogenous cirrhosis of the liver and in particular the role of ADH are considered. 3 cases out of 18 suffering from ascitic phase cirrhosis in whom inability to produce free water was accompanied by conserved urinary excretion of sodium are reported. One explanation might be the intervention of ADH or of an antidiuretic substance.
D000328:Adult; D000368:Aged; D001201:Ascites / Q000209:etiology*; D005260:Female; D006801:Humans; D007010:Hyponatremia / Q000209:etiology*; D008103:Liver Cirrhosis / Q000150:complications*; D008297:Male; D008875:Middle Aged; D009994:Osmolar Concentration; D014667:Vasopressins / Q000378:metabolism*; D014883:Water-Electrolyte Imbalance / Q000209:etiology
D014667:Vasopressins
La sindrome iposmolare-iponatriemica nella cirrosi epatica. Possibile ruolo dell'ADH.