[Adaptive and compensatory mechanisms in chronic circulatory insufficiency in ischemic heart disease].
Activation of some neuroendocrine systems is one of the compensatory-adaptation mechanisms in circulatory insufficiency in patients with ischemic heart disease. Evidence of this is that patients with stage I--IIA of the disease are marked by an increase in the activity of the sympatheticoadrenal system (increase in the excretion of free catecholamines) and the renin-angiotensin-aldosterone system, in the glucocorticoid function of the adrenal cortex, in the functional condition of the epiphysis cerebri, and in the blood plasma antidiuretic activity. Participation of these systems in the adaptation processes maintains an adequate level of venous blood return to the heart and inotropic myocardial function.
D000222:Adaptation, Physiological; D002302:Cardiac Output; D002318:Cardiovascular Diseases / Q000503:physiopathology*; D002908:Chronic Disease; D003327:Coronary Disease / Q000503:physiopathology*; D006706:Homeostasis; D006728:Hormones / Q000502:physiology; D006801:Humans; D006973:Hypertension / Q000503:physiopathology
D006728:Hormones
Nekotorye adaptatsionno-kompensatornye mekhanizmy pri khronicheskoĭ nedostaochnosti krovoobrashcheniia u bol'nykh ishemicheskoĭ bolezn'iu serdtsa.