Focal cerebellar hyperthermia: effects on cerebral paroxysmal afterdischarges.
Small area hyperthermia is used to selectively increase cerebellar activity, and, as shown by electroencephalographic tracings of sensorimotor area, focal cerebellar hyperthermia with temperatures limited between 39.5 degrees and 41 degrees C effectively reduces the duration of electrically induced afterdischarges. Additional observations on paroxysms induced in the caudate nucleus which involve the sensorimotor area indicate that these also show shortened durations during focal cerebellar hyperthermia. A comparison of effects on these two forebrain structures is given and some similarities are noted between these results and those reported from previous studies in which electrical stimuli were applied directly to the cerebellar cortex.
D000818:Animals; D002415:Cats; D002421:Caudate Nucleus / Q000503:physiopathology; D002525:Cerebellar Cortex / Q000503:physiopathology; D002529:Cerebellar Nuclei / Q000503:physiopathology; D002531:Cerebellum / Q000503:physiopathology*; D004292:Dominance, Cerebral / Q000502:physiology; D004558:Electric Stimulation; D004569:Electroencephalography; D005071:Evoked Potentials; D006358:Hot Temperature; D009434:Neural Pathways / Q000503:physiopathology; D012012:Red Nucleus / Q000503:physiopathology; D012640:Seizures / Q000503:physiopathology*; D013003:Somatosensory Cortex / Q000503:physiopathology; D013788:Thalamus / Q000503:physiopathology