The measurement and control of myocardial infarct size.
Direct, chemical, electrocardiographic and radio-isotopic methods are described for the estimation of myocardial infarct size in animals and man. Their relative points and failings are discussed. The effects of interventions, physical, metabolic and pharmacological, upon the size of myocardial infarcts, are examined and work attempting to reduce myocardial infarct size in man reviewed.
D000818:Animals; D004796:Clinical Enzyme Tests; D003326:Coronary Circulation; D003402:Creatine Kinase / Q000097:blood; D004562:Electrocardiography; D006801:Humans; D007770:L-Lactate Dehydrogenase / Q000097:blood; D009203:Myocardial Infarction / Q000175:diagnosis* / Q000503:physiopathology / Q000628:therapy*; D009206:Myocardium / Q000378:metabolism; D010101:Oxygen Consumption; D011877:Radionuclide Imaging; D000637:Transaminases / Q000097:blood
D007770:L-Lactate Dehydrogenase; D000637:Transaminases; D003402:Creatine Kinase