Palmitate and glucose oxidation by diaphragm of rats with fructose-induced hypertriglyceridemia.
Palmitate and glucose oxidation were investigated in isolated hemidiaphragm of rats in which hypertriglyceridemia was induced by dietary fructose. Palmitate oxidation was increased, and glucose oxidation was reduced, in fructose-fed rats, as compared with glucose-fed controls. Glucose incorporation into muscle glycogen was similar in both dietary groups.
D000818:Animals; D003964:Diaphragm / Q000378:metabolism; D004040:Dietary Carbohydrates; D005260:Female; D005632:Fructose; D005947:Glucose / Q000378:metabolism*; D006949:Hyperlipidemias / Q000139:chemically induced / Q000378:metabolism*; D009132:Muscles / Q000378:metabolism*; D010168:Palmitates / Q000378:metabolism*; D010169:Palmitic Acids / Q000378:metabolism*; D051381:Rats; D014280:Triglycerides / Q000097:blood*
D004040:Dietary Carbohydrates; D010168:Palmitates; D010169:Palmitic Acids; D014280:Triglycerides; D005632:Fructose; D005947:Glucose