[Acute hemorrhagic leucoencephalitis during tuberculosis (author's transl)].
The authors report a fairly typical clinico-pathological case of acute hemorragic leucoencephalitis (A.H.L.E.). Both clinical and histological features appeared particularly acute. At autopsy a visceral evolving tuberculosis was diagnosed. Such an etiological circumstance has not been, apparently, reported in cases of A.H.L.E. published as such. Three other etiological circumstances were noticed: pyuria, treatment by cephalotin and treatment by gentamycine. The physiopathogenesis of A.H.L.E. remains obscure and will not be clarified before detailed immunological studies can be performed. But the disease is rare and brisk and diagnosis usually post-mortem.
D001921:Brain / Q000473:pathology; D002512:Cephalothin / Q000009:adverse effects; D004679:Encephalomyelitis / Q000209:etiology*; D005260:Female; D005839:Gentamicins / Q000009:adverse effects; D006801:Humans; D004684:Leukoencephalitis, Acute Hemorrhagic / Q000139:chemically induced / Q000209:etiology* / Q000473:pathology; D008875:Middle Aged; D011776:Pyuria / Q000150:complications; D014398:Tuberculosis, Renal / Q000150:complications*
D005839:Gentamicins; D002512:Cephalothin
Leucoencéphalite aigue hémorragique au cours d'une tuberculose.