Peripheral circulation in arterial hypertension.
The underlying causes of hypertensive disease remain unclear. This article has attempted to highlight potential dysfunctions in arterial pressure regulation which could either initiate or sustain the hypertensive process. As has been suggested innumerable times hypertension must certainly be a multifactorial abnormality. The current state of knowledge about control of the peripheral circulation in high arterial pressure reinforces the view that interactions between neurogenic, myogenic, humoral, and structural factors are probably disrupted when hypertension appears.
D000804:Angiotensin II / Q000097:blood / Q000494:pharmacology; D000818:Animals; D001158:Arteries / Q000294:innervation / Q000503:physiopathology; D003097:Collateral Circulation / Q000187:drug effects; D006801:Humans; D006973:Hypertension / Q000503:physiopathology*; D006977:Hypertension, Renal / Q000097:blood; D006984:Hypertrophy / Q000503:physiopathology; D009119:Muscle Contraction; D009130:Muscle, Smooth / Q000503:physiopathology; D009489:Neurosecretion; D011311:Pressoreceptors / Q000503:physiopathology; D011453:Prostaglandins / Q000494:pharmacology; D012083:Renin / Q000097:blood; D014655:Vascular Resistance / Q000187:drug effects; D014666:Vasomotor System / Q000503:physiopathology
D011453:Prostaglandins; D000804:Angiotensin II; D012083:Renin