[Cardiac insufficiency in ischemic heart disease].
The presented analysis deals with the physiopathological mechanisms of the debelopment of postinfarction cardiac insufficiency and the clinical peculiarities of its manifestations. It is emphasized that the leading cause of cardiac insufficiency consists in a reduction of the contractile function of the left ventricular myocardium due to the development of asynergy in the cicatrical zone. The addition of several accompanying factors, such as heart mitralization, tachysystolic form of ventricular fibrillation, repeated infarction with a growing asynergic zone, aggravate the course of cardiac insufficiency providing for the development of hypertension in the general circulation system.
D000208:Acute Disease; D000222:Adaptation, Physiological; D001145:Arrhythmias, Cardiac / Q000503:physiopathology; D001810:Blood Volume; D002302:Cardiac Output; D002908:Chronic Disease; D003326:Coronary Circulation; D003327:Coronary Disease / Q000503:physiopathology*; D006321:Heart / Q000503:physiopathology; D006333:Heart Failure / Q000503:physiopathology; D006339:Heart Rate; D006346:Heart Septum / Q000503:physiopathology; D006352:Heart Ventricles / Q000503:physiopathology; D006801:Humans; D006973:Hypertension / Q000209:etiology; D009200:Myocardial Contraction; D009203:Myocardial Infarction / Q000503:physiopathology; D011654:Pulmonary Edema / Q000503:physiopathology; D012770:Shock, Cardiogenic / Q000503:physiopathology
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