[Pemphigus].
This review stresses particularly the role that "intercellular" antigens and antibodies, complement and lymphocytic dysfunction may play in pemphigus. In addition evidences in the literature in favour of exogenic factors inducing pemphigus are surveyed. Further an attempt is made to summarize the possibilities for the etiology and the blister formation in this disease.
D000906:Antibodies / Q000032:analysis; D000941:Antigens / Q000032:analysis; D001327:Autoimmune Diseases; D003165:Complement System Proteins; D004342:Drug Hypersensitivity; D006801:Humans; D007111:Immunity, Cellular; D066298:In Vitro Techniques; D010392:Pemphigus / Q000209:etiology* / Q000276:immunology / Q000473:pathology; D012867:Skin / Q000276:immunology / Q000473:pathology
D000906:Antibodies; D000941:Antigens; D003165:Complement System Proteins
Pemphiguskrankheiten