[Effect of vitamin E on the functional state of rat liver mitochondria following bilateral subdiaphragmatic vagotomy].
On the basis of a proven protective effect of vitamin "E" in regard to cellular metabolism in hypoxia and presuming that it is exactly the chronic hypoxia that forms the main pathogenetic link in the development of "deneravation changes" the effect of vitamin "E" on the function of hepatic metachondria in rats was studied polarographically and by using tetracycline, as a fluorescent probe, at different dates following bilateral subphrenic vagotomy. The available data prompted an inference of a marked stabilizing influence of the agent on the state of mitochondria in 7 and more days after vagotomy.
D000818:Animals; D002118:Calcium / Q000378:metabolism; D008099:Liver / Q000294:innervation*; D008297:Male; D008930:Mitochondria, Liver / Q000187:drug effects* / Q000378:metabolism; D010101:Oxygen Consumption; D051381:Rats; D013772:Tetramethylphenylenediamine / Q000378:metabolism; D014628:Vagotomy; D014810:Vitamin E / Q000494:pharmacology*
D014810:Vitamin E; D013772:Tetramethylphenylenediamine; D002118:Calcium
Deĭstvie vitamin E na funktsional'noe sostoianie mitokhondriĭ pecheni krys posle dvustoronneĭ poddiafragmal'noĭ vagotomii