[Hemostatic processes in aneurysmal hemorrhages].
Pathogenetic, diagnostic, and therapeutic problems and questions associated with ruptured cerebral aneurysms assume good knowledge of hemostatic processes. The three factors affecting hemostasia, namely, vasoactive, coagulative, and fibrinolytic risk factors in the blood, cerebro--spinal fluid, and at the site of damage to the vessel wall, are discussed withparticular reference to a thrombosing aneurysm. In the case of secondary hemorrhages it is necessary to determine the cause or pathogenesis, respectively, of the disturbance of coagulation or increase in fibrinolysis in order to be able to take suitable therapeutic measures and reduce the risk of secondary bleeding occurring within the first critical weeks after aneurysmal rupture.
D000933:Antifibrinolytic Agents / Q000008:administration & dosage; D001780:Blood Coagulation Tests; D001812:Blood-Brain Barrier / Q000187:drug effects; D002560:Cerebrovascular Circulation / Q000187:drug effects; D005342:Fibrinolysis / Q000187:drug effects; D006487:Hemostasis; D006801:Humans; D002532:Intracranial Aneurysm / Q000097:blood* / Q000175:diagnosis / Q000188:drug therapy; D002542:Intracranial Embolism and Thrombosis / Q000097:blood / Q000175:diagnosis / Q000188:drug therapy; D012008:Recurrence; D013345:Subarachnoid Hemorrhage / Q000097:blood* / Q000175:diagnosis / Q000188:drug therapy; D014662:Vasoconstrictor Agents / Q000008:administration & dosage
D000933:Antifibrinolytic Agents; D014662:Vasoconstrictor Agents
Psychiatrie, Neurologie und medizinische Psychologie Zeitschrift für die gesamte Nervenheilkunde und Psychotherapie