Proliferation of osteoclasts in rat bone following bleeding and femoral fractures.
There is a significant transient increase in the number of osteoclasts in intact bone after bleedings and fractures in rats. This rise in the osteoclast population might be due to an increased parathyroid activity released by the trauma, but other factors may be involved. Both bleedings and ractures in rats are followed by hypercalcemia.
D000818:Animals; D001842:Bone and Bones / Q000166:cytology*; D002118:Calcium / Q000097:blood; D002455:Cell Division; D005264:Femoral Fractures / Q000097:blood / Q000503:physiopathology*; D006400:Hematocrit; D006470:Hemorrhage / Q000097:blood / Q000503:physiopathology*; D010010:Osteoclasts / Q000502:physiology*; D010281:Parathyroid Hormone / Q000502:physiology; D051381:Rats; D012709:Serum Albumin / Q000378:metabolism
D010281:Parathyroid Hormone; D012709:Serum Albumin; D002118:Calcium