Pyridoxine deficiency and postnatal development of brain aspartate and alanine aminotransferase activities, and cholesterol levels in chicks.
Feeding a pyridoxine deficient diet, for 2 weeks after hatching, had no effect on post-hatching development of chick brain aspartate aminotransferase (L-aspartate:2-oxoglutarate aminotransferase, EC 2.6.1.1) activity or on cholesterol deposition in the brain, but significantly depressed the development of brain alanine aminotransferase (L-alanine:2-oxoglutarate aminotransferase, EC 2.5.1.2) activity. Feeding a pyridoxine deficient diet from 3 to 8 weeks of age had no effect on any of the three parameters studied.
D000410:Alanine Transaminase / Q000378:metabolism*; D000818:Animals; D001219:Aspartate Aminotransferases / Q000378:metabolism*; D001835:Body Weight; D001921:Brain / Q000033:anatomy & histology / Q000254:growth & development / Q000378:metabolism*; D002645:Chickens / Q000378:metabolism*; D002784:Cholesterol / Q000378:metabolism*; D008297:Male; D008954:Models, Biological; D009929:Organ Size; D013217:Starvation / Q000378:metabolism; D026681:Vitamin B 6 Deficiency / Q000378:metabolism*
D002784:Cholesterol; D001219:Aspartate Aminotransferases; D000410:Alanine Transaminase