[On the genetics of the delayed attack of substrates by strains of the Salmonella group (author's transl)].
The delayed attack of sugar alcohols by Salmonellae essentially goes back on a mutative event but doesnot represent a phenotypical or genotypical uniform phenomenon. The - frequently occuring - regularly split of sugars is caused by a simple looking fixed rate mutative event, and is characterized by an only moderate variation of the latent period preceding acid formation. In contrast, irregularly delayed splitting strains attack the substrate varying and, when occasion arises, with an extremely uncertain latency. Fermentation capacity of some strains attacking dulcitol irregularly can be induced by irradiation with ultraviolet light, and can be reduced to a certain degree by treatment with acridine orange, but cannot be transferred to other strains by conjugation. We presume that at least the uv inducible type of irregularly delayed splitting is governed by an episomal genetical element which is active only in the autonomous state but is repressed when being integrated within the chromosome.
D003470:Culture Media; D005285:Fermentation / Q000187:drug effects; D004376:Galactitol / Q000494:pharmacology; D009154:Mutation / Q000187:drug effects; D012475:Salmonella / Q000235:genetics*; D013997:Time Factors
D003470:Culture Media; D004376:Galactitol
Zur Genetik des verzögerten Substratangriffs bei Keimen der Salmonella-Gruppe.