Utilization of early promotors in mutant far P85 of bacteriophage T4.
We show that farP85 is a recessive mutant of T4 incapable of activating the delayed early promotors for genes 43 and 45 and that the farP85 mutation is in the same complementation group as the ts G1 mutation, which is located in the "modifier of transcription" (mot) gene.
D003090:Coliphages / Q000187:drug effects / Q000378:metabolism*; D004352:Drug Resistance, Microbial; D005492:Folic Acid / Q000031:analogs & derivatives / Q000494:pharmacology; D005796:Genes; D005816:Genetic Complementation Test; D009154:Mutation; D009876:Operon; D013696:Temperature; D014158:Transcription, Genetic; D014466:Ultraviolet Rays; D014764:Viral Proteins / Q000096:biosynthesis*
D014764:Viral Proteins; D005492:Folic Acid