Hemodynamic consequences of cardiac dysrhythmias.
To understand the hemodynamic changes produced by arrhythmias is to understand cardiovascular physiology. The changes in rate are accompanied by changes in contractility, receptor stimulation, peripheral vascular response, and flow to other vital organs. In addition, one must keep in mind the underlying pathology of the cardiovascular system. As a therapeutic program is initiated and adjusted, it must be done in accordance with the changes produced in the hemodynamics as well as the rhythm.
D000889:Anti-Arrhythmia Agents / Q000494:pharmacology; D001145:Arrhythmias, Cardiac / Q000150:complications / Q000188:drug therapy / Q000503:physiopathology*; D001281:Atrial Fibrillation / Q000503:physiopathology; D001282:Atrial Flutter / Q000503:physiopathology; D001794:Blood Pressure; D001919:Bradycardia / Q000503:physiopathology; D002302:Cardiac Output; D003326:Coronary Circulation; D004594:Electrophysiology; D006325:Heart Atria / Q000503:physiopathology; D006327:Heart Block / Q000503:physiopathology; D006339:Heart Rate; D006352:Heart Ventricles / Q000503:physiopathology; D006439:Hemodynamics; D006801:Humans; D012039:Regional Blood Flow; D013610:Tachycardia / Q000503:physiopathology
D000889:Anti-Arrhythmia Agents