Endocrine control of skeletal robusticity.
Changes of skeletal robusticity result from a mosaic response of various parts of the skeleton to given endocrine changes. The character of these reactions corresponds to that of muscles which act as selective target areas for specific hormones. Robusticity changes are mediated by increases or decreases of the length and weight of the bones. The changes are probably species- or even subspecies-specific.
D000818:Animals; D001846:Bone Development / Q000187:drug effects; D001842:Bone and Bones / Q000033:anatomy & histology* / Q000378:metabolism; D002369:Castration; D003864:Depression, Chemical; D004032:Diet; D004702:Endocrine Glands / Q000502:physiology*; D004967:Estrogens / Q000494:pharmacology; D005260:Female; D007016:Hypophysectomy; D008297:Male; D009929:Organ Size; D010053:Ovary / Q000502:physiology; D010902:Pituitary Gland / Q000502:physiology; D011374:Progesterone / Q000494:pharmacology; D051381:Rats; D012737:Sex Factors; D013268:Stimulation, Chemical; D013737:Testis / Q000502:physiology; D013739:Testosterone / Q000494:pharmacology; D013961:Thyroid Gland / Q000502:physiology; D013965:Thyroidectomy
D004967:Estrogens; D013739:Testosterone; D011374:Progesterone