[Granulocyte dysfunction. Part II. Secondary defects. (authors transl)].
In the first part we reviewed both the molecular basis of granulocyte function and the inborn defects. The present chapter summarizes our knowledge of the secondary defects of chemotaxis, opsonisation, phagocytosis and intracellular microbicidal activity.
D000818:Animals; D001172:Arthritis, Rheumatoid / Q000097:blood; D001424:Bacterial Infections / Q000097:blood; D002056:Burns / Q000097:blood; D002633:Chemotaxis; D002648:Child; D003920:Diabetes Mellitus / Q000097:blood; D064420:Drug-Related Side Effects and Adverse Reactions; D006098:Granulocytes / Q000473:pathology / Q000502:physiology / Q000528:radiation effects; D006168:Guinea Pigs; D006402:Hematologic Diseases / Q000097:blood / Q000209:etiology; D006801:Humans; D007674:Kidney Diseases / Q000097:blood; D007938:Leukemia / Q000097:blood; D007962:Leukocytes; D009748:Nutrition Disorders / Q000097:blood; D010587:Phagocytosis / Q000528:radiation effects; D011830:Radiation Effects
Granulocytendysfunktion. II. Erworbene Störungen