[Background activity of the neurons and brain tissue oxygen tension in asphyxia].
The activity level of the cerebral cortex as estimated by the unit activity was found to correlate with the PO2 parameters. Two phasic changes of the unit activity dueing asphyxia were confirmed. The initial firing rate was unrelated to decrease of the PO2 but to the reticular formation influence rather. The following reduction of the unit activity was connected with the PO2 decrease in the brain tissue.
D000818:Animals; D001237:Asphyxia / Q000503:physiopathology*; D001921:Brain / Q000503:physiopathology*; D002415:Cats; D002540:Cerebral Cortex / Q000503:physiopathology; D002746:Chlorpromazine / Q000494:pharmacology; D004594:Electrophysiology; D009474:Neurons / Q000502:physiology*; D010101:Oxygen Consumption; D010313:Partial Pressure; D011048:Polarography; D011941:Receptors, Adrenergic / Q000187:drug effects; D012154:Reticular Formation / Q000187:drug effects; D014793:Visual Cortex / Q000503:physiopathology
D011941:Receptors, Adrenergic; D002746:Chlorpromazine
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