[Features of hepatic lesions in patients with chronic alcoholic intoxication].
The authors studied alcoholic hepatosis and hepatic cirrhosis by laboratory, radioisotope and clinical methods. Most of the routine laboratory techniques, excluding hyperurobilinuria were not very informative in alcoholic hepatosis. Much more frequently it was possible to mark disturbances of the bromsulphaleinic and vofaverdine tests. Of special importance in the evaluation of the acuity and depth of the alcohol intoxication was glutamate and sorbitdehydrogenase. The most informative appeared to be radioisotope hepatography in the phase of alcohol hepatosis and scannography in the formation of liver cirrhosis.
D000410:Alanine Transaminase / Q000097:blood; D000429:Alcohol Oxidoreductases / Q000097:blood; D000435:Alcoholic Intoxication / Q000150:complications*; D001219:Aspartate Aminotransferases / Q000097:blood; D002908:Chronic Disease; D005969:Glutamate Dehydrogenase / Q000097:blood; D006801:Humans; D008103:Liver Cirrhosis / Q000175:diagnosis / Q000209:etiology; D008107:Liver Diseases / Q000175:diagnosis / Q000209:etiology*; D011877:Radionuclide Imaging; D013448:Sulfobromophthalein
D013448:Sulfobromophthalein; D000429:Alcohol Oxidoreductases; D005969:Glutamate Dehydrogenase; D001219:Aspartate Aminotransferases; D000410:Alanine Transaminase
Ob osobennostiakh povrezhdeniĭ pecheni u bol'nykh s khronicheskoĭ intoksikatsieĭ alkogolem