N-acetyl-cysteine is a potent suppressor of human immunodeficiency virus transcription in persistently infected cells.
Kinter
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Laboratory of Immunoregulation, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892.
Poli
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Fauci
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D000111:Acetylcysteine / Q000494:pharmacology*; D002460:Cell Line; D016325:HIV Long Terminal Repeat / Q000187:drug effects; D054303:HIV Reverse Transcriptase; D015497:HIV-1 / Q000187:drug effects* / Q000235:genetics / Q000502:physiology; D006801:Humans; D018894:Reverse Transcriptase Inhibitors; D013755:Tetradecanoylphorbol Acetate / Q000494:pharmacology; D014158:Transcription, Genetic / Q000187:drug effects; D014162:Transfection; D014409:Tumor Necrosis Factor-alpha / Q000494:pharmacology; D014779:Virus Replication / Q000187:drug effects
D018894:Reverse Transcriptase Inhibitors; D014409:Tumor Necrosis Factor-alpha; D054303:HIV Reverse Transcriptase; D013755:Tetradecanoylphorbol Acetate; D000111:Acetylcysteine