Signal transduction through small GTPases--a tale of two GAPs.
Hall
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Chester Beatty Laboratories, Institute of Cancer Research, London, England.
D000818:Animals; D004789:Enzyme Activation; D020558:GTP Phosphohydrolases / Q000502:physiology*; D020690:GTPase-Activating Proteins; D016514:Genes, Neurofibromatosis 1; D011905:Genes, ras; D006801:Humans; D025542:Neurofibromin 1; D010641:Phenotype; D019869:Phosphatidylinositol 3-Kinases; D010770:Phosphotransferases / Q000378:metabolism; D011506:Proteins / Q000737:chemistry / Q000502:physiology*; D016283:Proto-Oncogene Proteins p21(ras) / Q000378:metabolism*; D012689:Sequence Homology, Nucleic Acid; D015398:Signal Transduction; D020703:ras GTPase-Activating Proteins
D020690:GTPase-Activating Proteins; D025542:Neurofibromin 1; D011506:Proteins; D020703:ras GTPase-Activating Proteins; D010770:Phosphotransferases; D019869:Phosphatidylinositol 3-Kinases; D020558:GTP Phosphohydrolases; C501009:HRAS protein, human; D016283:Proto-Oncogene Proteins p21(ras)