Depression of burst discharges and of neuronal calcium influx by adenosine.
Schubert
P
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Department of Neuromorphology, Max Planck Institute for Psychiatry, Martinsried, Germany.
D000241:Adenosine / Q000502:physiology*; D000818:Animals; D002118:Calcium / Q000502:physiology*; D015220:Calcium Channels / Q000502:physiology*; D046508:Culture Techniques; D003712:Dendrites / Q000502:physiology; D004827:Epilepsy / Q000503:physiopathology*; D006624:Hippocampus / Q000503:physiopathology*; D009433:Neural Inhibition / Q000502:physiology*; D009474:Neurons / Q000502:physiology; D011188:Potassium / Q000502:physiology; D015221:Potassium Channels / Q000502:physiology; D011493:Protein Kinase C / Q000502:physiology; D051381:Rats; D011983:Receptors, Purinergic / Q000502:physiology; D013569:Synapses / Q000502:physiology; D013570:Synaptic Membranes / Q000502:physiology; D009435:Synaptic Transmission / Q000502:physiology*
D015220:Calcium Channels; D015221:Potassium Channels; D011983:Receptors, Purinergic; D011493:Protein Kinase C; D000241:Adenosine; D011188:Potassium; D002118:Calcium