Contribution of agonist-induced hyperpolarization to Ca2+ influx and formation of EDRF in vascular endothelial cells.
Kukovetz
W R
WR
Institut für Pharmakodynamik und Toxikologie, Universität Graz, Austria.
Graier
W F
WF
Groschner
K
K
D000818:Animals; D001920:Bradykinin / Q000494:pharmacology; D002118:Calcium / Q000378:metabolism*; D002120:Calcium Channel Agonists / Q000494:pharmacology*; D002478:Cells, Cultured; D006152:Cyclic GMP / Q000378:metabolism; D004594:Electrophysiology; D004730:Endothelium, Vascular / Q000187:drug effects / Q000378:metabolism*; D008564:Membrane Potentials / Q000187:drug effects; D009569:Nitric Oxide / Q000096:biosynthesis*; D015221:Potassium Channels / Q000187:drug effects; D000644:Quaternary Ammonium Compounds / Q000494:pharmacology; D013552:Swine
D002120:Calcium Channel Agonists; D015221:Potassium Channels; D000644:Quaternary Ammonium Compounds; D009569:Nitric Oxide; C009405:tetrabutylammonium; D006152:Cyclic GMP; D001920:Bradykinin; D002118:Calcium