[Physiopathology of atopic dermatitis].
This pathological sequence, although partly hypothetical, allows consideration of many points involved in therapy. These treatments arouse interest as to how they behave on the causal mechanisms. Thus, should symptomatic treatments that act on pruritus, xerosis, skin inflammation, of which the effect may be only transitory be offered, or active therapy on the initial factors which may have a much more radical effect on the illness?
Moureaux
P
P
D003876:Dermatitis, Atopic / Q000235:genetics / Q000276:immunology / Q000503:physiopathology* / Q000628:therapy; D005227:Fatty Acids / Q000378:metabolism; D006801:Humans; D007111:Immunity, Cellular / Q000187:drug effects; D007073:Immunoglobulin E / Q000276:immunology; D007801:Langerhans Cells / Q000276:immunology / Q000473:pathology; D008954:Models, Biological; D011943:Receptors, Adrenergic, beta / Q000502:physiology
D005227:Fatty Acids; D011943:Receptors, Adrenergic, beta; D007073:Immunoglobulin E
Physiopathologie de la dermatite atopique.