Decerebrate rigidity with preserved cognition and gait: a possible role of anoxic-ischemic brain damage.
A case of stable decerebrate posture in the upper limbs following sudden loss of consciousness and prolonged coma is described. The patient recovered most of her cognitive functions and gait, without clinical, neurophysiological or neuroradiological evidence of brainstem lesion. MRI shows borderzone infarcts. It is suggested that anoxic-ischemic cortical damage, affecting specially corticoreticular neurons, could explain the development of decerebrate rigidity in patients without apparent brainstem lesion.
Damasceno
B P
BP
Departamento de Neurologia, FCM, Universidade Estadual de Campinas, Brasil.
D000328:Adult; D001132:Arm / Q000503:physiopathology*; D001921:Brain / Q000000981:diagnostic imaging / Q000503:physiopathology; D002545:Brain Ischemia / Q000150:complications* / Q000503:physiopathology; D003655:Decerebrate State / Q000175:diagnosis / Q000209:etiology* / Q000503:physiopathology*; D005260:Female; D006801:Humans; D000860:Hypoxia / Q000150:complications* / Q000503:physiopathology; D008279:Magnetic Resonance Imaging; D009483:Neuropsychological Tests; D011859:Radiography