An in vitro study on increased neuronal and astrocytic vulnerability to neurotoxic injury after in utero cocaine exposure: the reversal effects of GM1 treatment.
Department of Anatomy, Chinese University of Hong Kong, N.T.
A
A
Stadlin
D
D
Tsang
J S
JS
MacDonall
S P
SP
Mahadik
S E
SE
Karpiak
D000014:Abnormalities, Drug-Induced / Q000517:prevention & control*; D000818:Animals; D000831:Animals, Newborn; D001253:Astrocytes / Q000187:drug effects*; D001692:Biological Transport / Q000187:drug effects; D001834:Body Water / Q000737:chemistry; D048429:Cell Size / Q000187:drug effects; D002478:Cells, Cultured; D002540:Cerebral Cortex / Q000002:abnormalities / Q000187:drug effects* / Q000196:embryology / Q000473:pathology; D003042:Cocaine / Q000494:pharmacology / Q000633:toxicity*; D005260:Female; D005315:Fetal Diseases / Q000139:chemically induced; D005677:G(M1) Ganglioside / Q000494:pharmacology / Q000627:therapeutic use*; D005971:Glutamates / Q000494:pharmacology; D018698:Glutamic Acid; D000860:Hypoxia / Q000139:chemically induced; D008431:Maternal-Fetal Exchange; D009474:Neurons / Q000187:drug effects*; D011188:Potassium / Q000494:pharmacology; D011247:Pregnancy; D051381:Rats; D017207:Rats, Sprague-Dawley
D005971:Glutamates; D005677:G(M1) Ganglioside; D018698:Glutamic Acid; D003042:Cocaine; D011188:Potassium