The role of GAP-43 in the molecular regulation of axon outgrowth and electrical excitability.
Baetge
E E
EE
Bristol-Myers Squibb Company, Pharmaceutical Research Institute, Wallingford, CT 06492.
Hammang
J P
JP
Gribkoff
V K
VK
Meiri
K F
KF
D000818:Animals; D001369:Axons / Q000502:physiology*; D015220:Calcium Channels / Q000502:physiology; D002448:Cell Adhesion; D003599:Cytoskeleton / Q000737:chemistry; D004594:Electrophysiology; D019922:GAP-43 Protein; D008562:Membrane Glycoproteins / Q000235:genetics / Q000502:physiology*; D008564:Membrane Potentials; D009419:Nerve Tissue Proteins / Q000235:genetics / Q000502:physiology*; D016501:Neurites / Q000502:physiology; D009469:Neuromuscular Junction; D016716:PC12 Cells / Q000378:metabolism / Q000648:ultrastructure; D010766:Phosphorylation; D011493:Protein Kinase C / Q000378:metabolism; D011499:Protein Processing, Post-Translational; D051381:Rats; D014162:Transfection
D015220:Calcium Channels; D019922:GAP-43 Protein; D008562:Membrane Glycoproteins; D009419:Nerve Tissue Proteins; D011493:Protein Kinase C