Inflammatory neuropathy: pathogenesis and clinical features.
The nature of the underlying mechanisms in inflammatory and immune-mediated neuropathies continues to represent an intensive area of research. Different auto-antibodies that are thought to cause specific neuropathic syndromes have been described. The involvement of T cells, cytokines, complement and class II molecules in the pathogenesis of these syndromes has also been studied. There is also intensive investigation into the area of immunotherapy, in particular in the use of intravenous immunoglobulin (Ig).
Steck
A J
AJ
Service de Neurologie, Centre Hospitalier Universitaire Vaudois, Lausanne, Switzerland.
D001323:Autoantibodies / Q000032:analysis; D003711:Demyelinating Diseases / Q000209:etiology* / Q000276:immunology; D004576:Electromyography; D005677:G(M1) Ganglioside / Q000276:immunology; D006801:Humans; D009185:Myelin Proteins / Q000276:immunology; D009460:Neurologic Examination; D010257:Paraneoplastic Syndromes / Q000209:etiology / Q000276:immunology; D011115:Polyneuropathies / Q000209:etiology* / Q000276:immunology; D011129:Polyradiculoneuropathy / Q000209:etiology / Q000276:immunology
D001323:Autoantibodies; D009185:Myelin Proteins; D005677:G(M1) Ganglioside