title: [The enzymatic activity of ammonia metabolism in the liver during the parenteral nitrogen feeding of animals with experimental liver failure].
abstract: It was established that liver insufficiency the activity of hepatic glutamate dehydrogenase is markedly reduced that is, apparently, the main cause of hyperammonemia that accompanies hepatic encephalopathy. Here occurs a significant reduction of the processes of ammonia detoxication with formation of glutamine in the liver as evidenced by changes of the activity of glutamine synthetase. Administration parenterally of a preparation of nitrous feeding increases essentially ammonia detoxicating function of muscular tissue which supplements the detoxicating function of the liver.
authors.forename: A G
authors.initials: AG
authors.lastname: Magarlamov
authors.forename: I V
authors.initials: IV
authors.lastname: Levchenko
authors.forename: M Iu
authors.initials: MIu
authors.lastname: Anoshina
authors.forename: O E
authors.initials: OE
authors.lastname: Ishchuk
mesh_terms: D000641:Ammonia / Q000378:metabolism*; D000818:Animals; D002252:Carbon Tetrachloride Poisoning / Q000150:complications; D008099:Liver / Q000201:enzymology*; D017093:Liver Failure / Q000139:chemically induced / Q000201:enzymology* / Q000628:therapy; D008297:Male; D010288:Parenteral Nutrition; D011492:Protein Hydrolysates / Q000008:administration & dosage*; D051381:Rats; D017208:Rats, Wistar
chemical_list: D011492:Protein Hydrolysates; D000641:Ammonia
vernacular_title: Aktivnost' nekotorykh fermentov obmena ammiaka v pecheni pri parenteral'nom azotistom pitanii zhivotnykh s éksperimental'noĭ pechenochnoĭ nedostatochnost'iu.