Visual processing deficits in dyslexia: receptors or neural mechanisms?
In 1989 and 1990 Grosser and Spafford attributed abnormalities in the peripheral vision of dyslexic subjects to an anomalous distribution of rods and cones. We argue that at the light levels used in these experiments cone responses should dominate both central and peripheral vision. A more likely explanation of their findings is that there is a postreceptoral deficit in the transient visual channel. This hypothesis is supported by independent anatomical, physiological, and psychophysical evidence.
Stuart
G W
GW
Centre for Information Science Research, Australian National University, Victoria.
Lovegrove
W J
WJ
D001288:Attention / Q000502:physiology; D002648:Child; D004410:Dyslexia / Q000503:physiopathology*; D006801:Humans; D010786:Photoreceptor Cells / Q000503:physiopathology*; D014794:Visual Fields / Q000502:physiology*; D014795:Visual Pathways / Q000502:physiology; D014796:Visual Perception / Q000502:physiology*