Albert Einstein's dyslexia and the significance of Brodmann Area 39 of his left cerebral cortex.
By his own admission, Albert Einstein, 'started to talk comparatively late ... certainly not younger than three', and also had 'poor memory of words', during his childhood years. If lesions in Brodmann Area 39 of the cerebral hemisphere results in dyslexia, the 1985 report on the study of Einstein's brain that the neuron:glial ratio of Area 39 in the left cerebral hemisphere of the physicist was significantly smaller than that of the control values, provides a neuroanatomical clue to Einstein's childhood dyslexia. Though not discrediting this finding, some questions are raised in this paper regarding the controls employed in this 1985 report (1).
Kantha
S S
SS
Osaka BioScience Institute, Japan.
D002540:Cerebral Cortex / Q000254:growth & development / Q000473:pathology*; D004410:Dyslexia / Q000266:history* / Q000473:pathology; D049672:History, 19th Century; D049673:History, 20th Century; D006801:Humans; D008297:Male; D010825:Physics / Q000266:history*