Acute methanol poisoning: a case study.
Acute methanol poisoning produces severe anion gap metabolic acidosis caused by the toxic accumulation of metabolites, primarily formic acid. Formic acid produces serious neurologic sequelae. Therefore, an understanding of the mechanism of toxicity, treatment, and clinical course is essential in preventing permanent neurologic dysfunction. Prompt recognition and treatment are the keys to successful patient outcomes. This article presents a case report of a patient with severe methanol poisoning with clinical course, treatment, and outcome.
King
L
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School of Nursing, University of California, Los Angeles.
D000328:Adult; D004630:Emergencies; D000431:Ethanol / Q000627:therapeutic use*; D006801:Humans; D008140:Lorazepam / Q000627:therapeutic use; D008297:Male; D000432:Methanol / Q000378:metabolism / Q000506:poisoning*; D009994:Osmolar Concentration; D011015:Pneumonia, Aspiration / Q000209:etiology; D012121:Respiration, Artificial; D012640:Seizures / Q000139:chemically induced / Q000188:drug therapy
D000431:Ethanol; D008140:Lorazepam; D000432:Methanol