[Microvascular reactivity and transcapillary fluid exchange in the lungs].
In anesthetized cats and rabbits responses of terminal pulmonary vessels to alveolar hypoxia, noradrenaline, histamin, acetylcholine and serotonine, were studied. The role of the vascular bed tone in regulation of pulmonary filtration-absorption function, was studied. A low motor activity of pulmonary microvessels was found. Their contribution to hemodynamic shifts in microcirculation was insignificant. Pulmonary vasomotor responses cannot be a reason for considerable extravascular accumulation of fluid in the lungs at a normal hydraulic permeability of alveolar-capillary membrane.
Dvoretskiĭ
D P
DP
D000042:Absorption; D000818:Animals; D002196:Capillaries / Q000187:drug effects / Q000502:physiology; D002199:Capillary Permeability / Q000187:drug effects / Q000502:physiology; D002415:Cats; D015633:Extravascular Lung Water / Q000187:drug effects / Q000502:physiology*; D006874:Hydrostatic Pressure; D000860:Hypoxia / Q000503:physiopathology; D008168:Lung / Q000098:blood supply* / Q000187:drug effects; D008833:Microcirculation / Q000187:drug effects / Q000502:physiology; D008853:Microscopy / Q000295:instrumentation / Q000379:methods; D011650:Pulmonary Alveoli / Q000187:drug effects / Q000502:physiology; D011817:Rabbits; D014662:Vasoconstrictor Agents / Q000494:pharmacology; D014665:Vasodilator Agents / Q000494:pharmacology
D014662:Vasoconstrictor Agents; D014665:Vasodilator Agents
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