DL-threo-3-hydroxyaspartate reduces NMDA receptor activation by glutamate in cultured neurons.
Marini
A
A
Clinical Neuroscience Branch, National Institute for Neurological Disorders and Stroke (NINDS), Bethesda, MD 20892.
Novelli
A
A
D000818:Animals; D001224:Aspartic Acid / Q000031:analogs & derivatives / Q000494:pharmacology; D002478:Cells, Cultured; D002531:Cerebellum / Q000166:cytology / Q000187:drug effects / Q000378:metabolism; D006152:Cyclic GMP / Q000096:biosynthesis; D005971:Glutamates / Q000494:pharmacology*; D018698:Glutamic Acid; D007608:Kainic Acid / Q000494:pharmacology; D009474:Neurons / Q000187:drug effects / Q000378:metabolism*; D051381:Rats; D016194:Receptors, N-Methyl-D-Aspartate / Q000187:drug effects*
D005971:Glutamates; D016194:Receptors, N-Methyl-D-Aspartate; C011274:3-hydroxyaspartic acid; D001224:Aspartic Acid; D018698:Glutamic Acid; D006152:Cyclic GMP; D007608:Kainic Acid