[Biochemical characteristics of uncomplicated and complicated healing of experimental myocardial infarcts].
Myocardial infarction was simulated in experiments with 138 dogs showing normal, high, and low responses. The kinetics of creatine kinase as a marker of necrotic processes in myocardial infarction was shown to accelerate with hyperreactivity and to slow down with hyporeactivity. The kinetics of ů-glutamyl transpeptidase as a marker of reparative processes in myocardial infarction was characterized by delayed and slower evolution in hyper- and hyporeactivity. The kinetics of dienic conjugates, cyclic nucleotides (cAMP, cGMP) and immunoreactive insulin was also determined largely by the body's reactivity and the types of experimental myocardial infarction healing and was closely related to that of enzymatic markers of necrotic processes.
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Iabluchanskiĭ
D000324:Adrenocorticotropic Hormone / Q000097:blood; D000818:Animals; D003402:Creatine Kinase / Q000097:blood; D004285:Dogs; D004837:Epinephrine / Q000097:blood; D006854:Hydrocortisone / Q000097:blood; D007328:Insulin / Q000097:blood; D007962:Leukocytes / Q000378:metabolism; D009203:Myocardial Infarction / Q000097:blood* / Q000150:complications; D009211:Myoglobin / Q000097:blood; D009712:Nucleotides, Cyclic / Q000097:blood; D005723:gamma-Glutamyltransferase / Q000097:blood
D007328:Insulin; D009211:Myoglobin; D009712:Nucleotides, Cyclic; D000324:Adrenocorticotropic Hormone; D005723:gamma-Glutamyltransferase; D003402:Creatine Kinase; D006854:Hydrocortisone; D004837:Epinephrine
Biokhimicheskaia kharakteristika neoslozhnennogo i oslozhnennykh form zazhivlenia éksperimental'nogo infarkta miokarda.