Reoxygenation of the hypoxic myocardium causes a mitochondrial complex I defect.
Department of Biochemistry, St. Bartholomew's Hospital Medical College, London, U.K.
D L
DL
Hardy
J B
JB
Clark
V M
VM
Darley-Usmar
D R
DR
Smith
D000818:Animals; D002118:Calcium / Q000378:metabolism; D004579:Electron Transport; D005182:Flavin-Adenine Dinucleotide / Q000378:metabolism; D066298:In Vitro Techniques; D008929:Mitochondria, Heart / Q000187:drug effects / Q000378:metabolism*; D015428:Myocardial Reperfusion Injury / Q000378:metabolism*; D009243:NAD / Q000378:metabolism; D016660:NAD(P)H Dehydrogenase (Quinone); D011808:Quinone Reductases / Q000378:metabolism*; D051381:Rats; D012430:Ruthenium Red / Q000494:pharmacology
D009243:NAD; D012430:Ruthenium Red; D005182:Flavin-Adenine Dinucleotide; D016660:NAD(P)H Dehydrogenase (Quinone); D011808:Quinone Reductases; D002118:Calcium