Hypothesis: antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination.
Cross
A H
AH
Department of Neurology, Albert Einstein College of Medicine, Bronx, NY 10461.
Cannella
B
B
Brosnan
C F
CF
Raine
C S
CS
D000818:Animals; D000941:Antigens / Q000276:immunology*; D001327:Autoimmune Diseases / Q000473:pathology / Q000503:physiopathology*; D002490:Central Nervous System / Q000098:blood supply*; D002493:Central Nervous System Diseases / Q000473:pathology / Q000503:physiopathology*; D003711:Demyelinating Diseases / Q000473:pathology / Q000503:physiopathology*; D004730:Endothelium, Vascular / Q000503:physiopathology*; D000939:Epitopes; D007249:Inflammation / Q000473:pathology / Q000503:physiopathology; D051379:Mice; D013601:T-Lymphocytes / Q000276:immunology / Q000502:physiology*
D000941:Antigens; D000939:Epitopes