[Pathogenesis of secondary glaucoma in epithelial invasion].
Apart from meticulous wound closure certain amount of fibrin and fibroblastic activity are necessary to prevent epithelial ingrowth. Prostaglandin inhibitors and steroid may enhance such danger. The most frequent reason for glaucoma were peripheral anterior synechiae. Open angle glaucoma with stratified epithelium lining the trabeculum was less frequent. Glaucoma can be caused by goblet cells produced mucus blocking the trabeculum. Desquamated epithelial cells, pupillary block caused by epithelial invasion of anterior hyaloid and intense inflammatory reaction can also exceptionally induce secondary glaucoma.
Zagórski
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Z
Kliniki Okulistycznej Uniwersytetu w Erlangen-Norymberdze, RFN.
Lang
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Naumann
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G
D002455:Cell Division / Q000502:physiology; D003315:Cornea / Q000473:pathology; D065306:Corneal Injuries; D004728:Endothelium, Corneal / Q000473:pathology*; D005347:Fibroblasts / Q000473:pathology; D005902:Glaucoma, Open-Angle / Q000209:etiology* / Q000473:pathology; D006801:Humans; D000267:Tissue Adhesions / Q000150:complications / Q000473:pathology; D014129:Trabecular Meshwork / Q000473:pathology*
Patogeneza jaskry wtórnej w inwazji nabłonka.