[Causes, sequelae and possible ways of preventing hypophosphatemia in patients with cerebral ischemia].
Hypophosphatemia revealed in patients with acute disorders of cerebral circulation was one of the causes of a decrease of the content of 2,3-DPG, ATP in red blood cells and of a reduction of Ca2(+)-ATPase activity in red blood cell membranes. In the given group of patients, hypophosphatemia was provoked by complete parenteral feeding, hypocapnia and by loss of phosphorus with urine and congestive gastrointestinal contents.
Pas'ko
S A
SA
Volosheniuk
T G
TG
D019794:2,3-Diphosphoglycerate; D000208:Acute Disease; D000255:Adenosine Triphosphate / Q000097:blood; D002545:Brain Ischemia / Q000097:blood* / Q000150:complications; D000252:Calcium-Transporting ATPases / Q000097:blood* / Q000172:deficiency; D004163:Diphosphoglyceric Acids / Q000097:blood; D004910:Erythrocyte Membrane / Q000201:enzymology*; D004912:Erythrocytes / Q000378:metabolism*; D006801:Humans; D010710:Phosphates / Q000008:administration & dosage / Q000097:blood* / Q000172:deficiency; D010760:Phosphorus Metabolism Disorders / Q000209:etiology* / Q000517:prevention & control
D004163:Diphosphoglyceric Acids; D010710:Phosphates; D019794:2,3-Diphosphoglycerate; D000255:Adenosine Triphosphate; D000252:Calcium-Transporting ATPases
Prichiny, sledstviia i vozmozhnye puti preduprezhdeniia gipofosfa- temii u bol'nykh s tserebral'nymi disgemiiami.