Glucocorticoids, hippocampal damage and the glutamatergic synapse.
Sapolsky
R M
RM
Department of Biological Sciences, Stanford University, CA 94305.
D000375:Aging / Q000502:physiology*; D000818:Animals; D015220:Calcium Channels / Q000187:drug effects; D002470:Cell Survival / Q000187:drug effects; D003071:Cognition / Q000187:drug effects; D004357:Drug Synergism; D004734:Energy Metabolism / Q000187:drug effects*; D005938:Glucocorticoids / Q000009:adverse effects / Q000494:pharmacology* / Q000502:physiology; D005971:Glutamates / Q000502:physiology*; D018698:Glutamic Acid; D006624:Hippocampus / Q000187:drug effects* / Q000378:metabolism; D015640:Ion Channel Gating / Q000187:drug effects; D008297:Male; D008954:Models, Biological; D009474:Neurons / Q000187:drug effects; D009498:Neurotoxins / Q000494:pharmacology; D051381:Rats; D016194:Receptors, N-Methyl-D-Aspartate / Q000187:drug effects; D015398:Signal Transduction / Q000187:drug effects; D013312:Stress, Physiological / Q000503:physiopathology*; D013569:Synapses / Q000187:drug effects* / Q000378:metabolism
D015220:Calcium Channels; D005938:Glucocorticoids; D005971:Glutamates; D009498:Neurotoxins; D016194:Receptors, N-Methyl-D-Aspartate; D018698:Glutamic Acid