[Dissecting aortic aneurysm associated with myonephropathic-metabolic syndrome and hypercalcemia].
A forty-two years old male underwent an aortic arch replacement for an emergency treatment of dissecting aortic aneurysm (DeBakey type I). Separate cardiopulmonary bypass was used with main arterial inflow cannula inserted to right femoral artery. After the operation, ischemia of the right lower extremity led to acute renal failure due to myonephropathic-metabolic syndrome. Peritoneal dialysis, hemodialysis, and continuous arterio-venous hemofiltration were performed. Renal failure improved gradually. At the diuretic phase serum calcium concentration began to rise. Inspite of large amount of fluid and furosemide injection it became higher and finally reached to 20 mg/dl level. Calcitonin injection (320 mu/day) was very effective. In 2 months after surgery serum creatinine and calcium concentrations went down to normal range. Abnormalities in calcium metabolism are frequent in rhabdomyolysis-induced acute renal failure. However, it is rare to encounter such a remarkable hypercalcemia as seen in this patient. When treating MNMS we should pay attention to the changes of serum calcium concentration.
Uchida
K
K
First Department of Surgery, Yokohama City University School of Medicine, Japan.
Kondo
J
J
Imoto
K
K
Kajiwara
H
H
Mashimo
Y
Y
Matsumoto
A
A
D058186:Acute Kidney Injury / Q000209:etiology*; D000328:Adult; D000784:Aortic Dissection / Q000601:surgery*; D001013:Aorta, Thoracic / Q000601:surgery; D001014:Aortic Aneurysm / Q000601:surgery*; D001807:Blood Vessel Prosthesis; D006801:Humans; D006934:Hypercalcemia / Q000209:etiology*; D008297:Male; D011183:Postoperative Complications; D012206:Rhabdomyolysis / Q000209:etiology*; D013577:Syndrome