[Pulmonary hypertension and myocardial contractility in myocarditis].
Hemodynamic studies by direct and indirect methods in patients with myocarditis have revealed that as pulmonary hypertension developed, right-ventricular myocardial contractility and pulmonary arteriolar bed elasticity were decreasing. High pulmonary hypertension and structural shifts in pulmonary artery wall reduced the volumic potentialities of the lesser circulation. The antiorthostatic test was found an important test for the detection of compensatory deposition potentialities of the lesser circulation vessels in myocarditis patients.
Gurevich
M A
MA
Baklykova
S N
SN
Novoderezhkina
L B
LB
Mravian
S R
SR
D000293:Adolescent; D000328:Adult; D002303:Cardiac Output, Low / Q000209:etiology*; D005260:Female; D006439:Hemodynamics / Q000502:physiology; D006801:Humans; D006976:Hypertension, Pulmonary / Q000209:etiology*; D008297:Male; D008875:Middle Aged; D009200:Myocardial Contraction / Q000502:physiology*; D009205:Myocarditis / Q000150:complications / Q000503:physiopathology*; D011652:Pulmonary Circulation / Q000502:physiology*; D016278:Ventricular Function, Right / Q000502:physiology*
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