[Dilated myocarditis and cardiomyopathies. The dysimmune hypothesis].
A causal relationship between myocarditis and idiopathic dilated cardiomyopathy has long been suspected. Humoral and cellular immune mechanisms are most probably involved in the pathogenesis of these diseases. Human genotype may influence the outcome of lymphocytic myocarditis by means of the major histocompatibility complex.
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Service de médecine interne et maladies cardio-vasculaires, Hôpital Avicenne, Bobigny.
D000818:Animals; D001327:Autoimmune Diseases / Q000209:etiology / Q000235:genetics / Q000276:immunology*; D002311:Cardiomyopathy, Dilated / Q000175:diagnosis / Q000209:etiology / Q000235:genetics / Q000276:immunology*; D005802:Genes, MHC Class II / Q000276:immunology; D006801:Humans; D051379:Mice; D008807:Mice, Inbred BALB C; D008955:Models, Cardiovascular; D009205:Myocarditis / Q000175:diagnosis / Q000209:etiology / Q000235:genetics / Q000276:immunology*; D051381:Rats; D013601:T-Lymphocytes / Q000276:immunology; D014777:Virus Diseases / Q000150:complications
Myocardites et cardiomyopathies dilatées. La conception dysimmunitaire.