title: [Supplementary concepts in the physiopathology of the vitreous].
abstract: The normal vitreous defends the posterior pole damping the traumatic and tension shocks coming from the anterior pole reducing their intensity according to formula P = F/S The buffering function decreases in direct proportion to the degree of fluidity of the vitreous, fluidity that enlarges the volume of the vitreous whereas the mass does not change according to the density formula p = m/v, the density of the liquefied vitreous being greatly diminished. The liquefied vitreous may lead to degenerative lesions of the retina due to prolonged tension of the retina resulting in ruptures and detachment. An anterior pole pressure of 15-16 mmHg transmitted non-modified to the posterior pole through the liquefied vitreous, exceeding by far the pressure of the opticoretinal tissular fluid pressure which in 9 mmHg leads to papillary excavation and glaucoma without tension; hence the liquefied vitreous must be replaced by a normal one or its density built up again.
authors.affiliation: Serviciul Oftalmologic, Policlinica nr. 1, Craiova.
authors.forename: D C
authors.initials: DC
authors.lastname: Begu
authors.forename: M R
authors.initials: MR
authors.lastname: Mănescu
mesh_terms: D005128:Eye Diseases / Q000503:physiopathology; D006801:Humans; D007429:Intraocular Pressure / Q000502:physiology; D009900:Optic Nerve / Q000503:physiopathology; D012160:Retina / Q000503:physiopathology; D014822:Vitreous Body / Q000503:physiopathology*
vernacular_title: Adaosuri conceptuale în fiziopathologia vitrosului.