title: Possible presence of V1 vasopressin receptor in toad urinary bladder.
abstract: The V1 vasopressin receptor antagonist, d(CH2)5Try(Me)AVP, enhanced the vasopressin-stimulated osmotic water flow in the toad bladder and inhibited the vasopressin-induced prostaglandin E2 production at 1 x 10(-9) M. It is well known that prostaglandin E2 inhibits the vasopressin-induced water flow in the toad bladder. These results indicate that the V1 vasopressin receptor is possibly present in the toad urinary bladder.
authors: Department of Physiology, Kitasato University School of Medicine, Kanagawa.
authors: Y
authors: Y
authors: Nara
authors: F
authors: F
authors: Marumo
mesh_terms: D000818:Animals; D001127:Arginine Vasopressin / Q000031:analogs & derivatives; D002023:Bufo bufo; D015232:Dinoprostone / Q000378:metabolism; D011945:Receptors, Angiotensin / Q000032:analysis*; D017483:Receptors, Vasopressin; D001743:Urinary Bladder / Q000032:analysis*; D014667:Vasopressins / Q000032:analysis*; D014882:Water-Electrolyte Balance
chemical_list: D011945:Receptors, Angiotensin; D017483:Receptors, Vasopressin; D014667:Vasopressins; D001127:Arginine Vasopressin; D015232:Dinoprostone