[Pathogenetic therapy of hemorrhagic strokes].
The authors provide grounds for the principles of pathogenic therapy aimed at posthemorrhagic cerebral vasospasm, proliferation-necrotic angiopathy with ensuing cerebral damage wherein free radical oxidation and antifibrinolytic therapy are of major importance. The efficiency of combined pathogenic therapy comprising antioxidants, chelators, cyclooxygenase inhibitors, calcium antagonists, barbiturate protectors, membrane-stabilizing and limiting antifibrinolytic hemostatic drugs is evaluated.
Gabashvili
V M
VM
Skakarishvili
R R
RR
Dzhanelidze
M T
MT
Dzhandzhgava
T V
TV
Maruashvili
M G
MG
D000933:Antifibrinolytic Agents / Q000008:administration & dosage; D000975:Antioxidants / Q000008:administration & dosage; D001921:Brain / Q000098:blood supply* / Q000378:metabolism; D002121:Calcium Channel Blockers / Q000008:administration & dosage; D002543:Cerebral Hemorrhage / Q000150:complications* / Q000188:drug therapy; D002544:Cerebral Infarction / Q000188:drug therapy* / Q000209:etiology / Q000378:metabolism; D016861:Cyclooxygenase Inhibitors; D004359:Drug Therapy, Combination; D005609:Free Radicals; D006490:Hemostatics / Q000008:administration & dosage; D006801:Humans; D011451:Prostaglandin-Endoperoxide Synthases / Q000378:metabolism
D000933:Antifibrinolytic Agents; D000975:Antioxidants; D002121:Calcium Channel Blockers; D016861:Cyclooxygenase Inhibitors; D005609:Free Radicals; D006490:Hemostatics; D011451:Prostaglandin-Endoperoxide Synthases
Patogeneticheskaia terapiia gemorragicheskikh insul'tov.