[Changes in the levels of somatotropin and cyclic nucleotides and the state of protein metabolism in the brain and myocardium during stress as well as in myocardial necrosis following stress].
Stress itself as well as the subsequent myocardium necrosis developed after stress were mainly responsible for distinct decrease in concentration of somatotropin, somatomedin and for impairments in protein synthesis accompanied by decrease in proteins half-life in brain hemispheres and in non-ischemized heart left ventricle.
Iakushev
V S
VS
Kuripka
V I
VI
Belokon'
L E
LE
Mironova
E V
EV
Krisanova
N V
NV
Davydov
V V
VV
Skurygin
V P
VP
D000818:Animals; D001921:Brain / Q000378:metabolism*; D000242:Cyclic AMP / Q000097:blood / Q000378:metabolism; D006152:Cyclic GMP / Q000097:blood / Q000378:metabolism; D013006:Growth Hormone / Q000097:blood / Q000378:metabolism; D008297:Male; D009203:Myocardial Infarction / Q000209:etiology / Q000378:metabolism*; D009206:Myocardium / Q000378:metabolism*; D014176:Protein Biosynthesis; D051381:Rats; D011919:Rats, Inbred Strains; D013315:Stress, Psychological / Q000150:complications / Q000378:metabolism*; D013997:Time Factors
D013006:Growth Hormone; D000242:Cyclic AMP; D006152:Cyclic GMP
Izmenenie kontsentratsii somatotropina, tsiklicheskikh nukleotidov i sostoianie belkovogo obmena v mozge i serdtse pri stresse, a takzhe nekroze miokarda, vosproizvedennom posle perenesennogo stressa.