title: The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase.
authors.forename: S B
authors.initials: SB
authors.lastname: Shears
authors.forename: A P
authors.initials: AP
authors.lastname: Dawson
authors.forename: J W
authors.initials: JW
authors.lastname: Loomis-Husselbee
authors.forename: P J
authors.initials: PJ
authors.lastname: Cullen
mesh_terms: D000535:Aluminum / Q000494:pharmacology*; D000818:Animals; D002118:Calcium / Q000378:metabolism*; D066298:In Vitro Techniques; D015544:Inositol 1,4,5-Trisphosphate / Q000378:metabolism*; D000072181:Inositol Polyphosphate 5-Phosphatases; D008862:Microsomes, Liver / Q000378:metabolism*; D010744:Phosphoric Monoester Hydrolases / Q000378:metabolism; D010770:Phosphotransferases / Q000378:metabolism; D017853:Phosphotransferases (Alcohol Group Acceptor); D051381:Rats; D015398:Signal Transduction
chemical_list: D015544:Inositol 1,4,5-Trisphosphate; D000535:Aluminum; D010770:Phosphotransferases; D017853:Phosphotransferases (Alcohol Group Acceptor); C051038:Inositol 1,4,5-trisphosphate 3-kinase; D010744:Phosphoric Monoester Hydrolases; D000072181:Inositol Polyphosphate 5-Phosphatases; D002118:Calcium