Activation of K+ channel and inhibition of Na(+)-K+ ATPase of human erythrocytes by cyclosporine: possible role in hyperpotassemia in kidney transplant recipients.
Ihara
H
H
Department of Urology, Hyogo College of Medicine, Japan.
Hosokawa
S
S
Ogino
T
T
Arima
M
M
Ikoma
F
F
D003524:Cyclosporins / Q000494:pharmacology*; D004910:Erythrocyte Membrane / Q000201:enzymology*; D004912:Erythrocytes / Q000187:drug effects / Q000378:metabolism*; D006801:Humans; D016030:Kidney Transplantation / Q000502:physiology*; D007700:Kinetics; D011188:Potassium / Q000097:blood; D015221:Potassium Channels / Q000187:drug effects / Q000502:physiology*; D012016:Reference Values; D012964:Sodium / Q000097:blood; D000254:Sodium-Potassium-Exchanging ATPase / Q000037:antagonists & inhibitors / Q000097:blood*
D003524:Cyclosporins; D015221:Potassium Channels; D012964:Sodium; D000254:Sodium-Potassium-Exchanging ATPase; D011188:Potassium