[A case of cranial polyneuropathy presenting with prominent facial diplegia, elevated serum Borrelia burgdorferi antibody and lymphocytic pleocytosis].
A 54 year-old male patient developed acute cranial polyneuropathy including prominent facial diplegia and radicular++-neuritis. He was proven to have lymphocytic pleocytosis, and elevated serum Borrelia burgdorferi antibody to X800 (normal; less than X200). A diagnosis of typical early neuro-borreliosis was made after these clinical and laboratory findings. This case is the first neuro-borreliosis showing the triad of neurological manifestations (meningitis, cranial neuritis, radicular++-neuritis) in Japan. It is concluded that neuro-borreliosis should be considered to be a cause of acute cranial polyneuropathy, particularly of facial diplegia, even if the patient has no apparent history of a tick bite.
Hasegawa
Y
Y
Department of Neurology, Nagoya National Hospital.
Ueno
S
S
Takegami
T
T
Okamoto
S
S
D000907:Antibodies, Bacterial / Q000032:analysis; D015748:Borrelia burgdorferi Group / Q000276:immunology*; D002555:Cerebrospinal Fluid / Q000166:cytology*; D003389:Cranial Nerve Diseases / Q000209:etiology*; D005158:Facial Paralysis / Q000209:etiology*; D006801:Humans; D008193:Lyme Disease / Q000150:complications*; D008218:Lymphocytosis / Q000209:etiology*; D008297:Male; D008875:Middle Aged; D011843:Radiculopathy / Q000209:etiology
D000907:Antibodies, Bacterial