[Characteristics of disorders of hemostasis and lipid metabolism in patients with different pathogenetic forms of angina pectoris].
The likely mechanisms responsible for the occurrence of angina pectoris, which were revealed by 24-hour ECG monitoring, were correlated with abnormalities in hemostasis and blood lipid spectrum. Changes in the blood lipid spectrum were demonstrated to play the leading role in the development of angina due to higher myocardial oxygen demand. Those in platelet hemostasis are mostly pronounced in impaired myocardial oxygen supply due to vessel spasm and/or formation of platelet aggregates.
Orlov
V N
VN
Vasil'eva
E Iu
EIu
Shpektor
A V
AV
Raskurazhev
A B
AB
Lekhormakher
S S
SS
D000328:Adult; D000368:Aged; D000787:Angina Pectoris / Q000097:blood* / Q000378:metabolism; D002784:Cholesterol / Q000097:blood; D008076:Cholesterol, HDL / Q000097:blood; D008078:Cholesterol, LDL / Q000097:blood; D005260:Female; D005340:Fibrinogen / Q000032:analysis; D006400:Hematocrit; D006487:Hemostasis; D006801:Humans; D050356:Lipid Metabolism; D008055:Lipids / Q000097:blood; D008297:Male; D008875:Middle Aged; D010974:Platelet Aggregation; D014280:Triglycerides / Q000097:blood; D014841:von Willebrand Factor / Q000032:analysis
D008076:Cholesterol, HDL; D008078:Cholesterol, LDL; D008055:Lipids; D014280:Triglycerides; D014841:von Willebrand Factor; D005340:Fibrinogen; D002784:Cholesterol
Osobennosti narusheniia gemostaza i lipidnogo obmena u bol'nykh s razlichnymi patogeneticheskimi formami stenokardii.