title: [Metabolism and septic shock].
abstract: Despite continuous monitoring progress, septic shock remains the main cause of death in Intensive Care Units. Septic stress triggers a complex array of biological modifications resulting in an acquired disease of intermediary metabolism whose main characteristic is an inefficient cellular use of glucidic, lipidic and protidic substrates together with oxygen. The purpose of this review is to present metabolic disturbances observed during sepsis and septic shock.
authors.affiliation: Laboratoire de Biochimie, Hôpital Tenon, Paris, France.
authors.forename: G
authors.initials: G
authors.lastname: Lefèvre
authors.forename: J F
authors.initials: JF
authors.lastname: Dhainaut
authors.forename: F
authors.initials: F
authors.lastname: Tallet
authors.forename: D
authors.initials: D
authors.lastname: Raichvarg
mesh_terms: D001424:Bacterial Infections / Q000378:metabolism*; D005960:Glucosides / Q000378:metabolism; D006019:Glycolysis; D006801:Humans; D050356:Lipid Metabolism; D009124:Muscle Proteins / Q000378:metabolism; D010100:Oxygen / Q000378:metabolism; D012772:Shock, Septic / Q000378:metabolism*
chemical_list: D005960:Glucosides; D009124:Muscle Proteins; D010100:Oxygen
vernacular_title: Métabolisme et choc septique.