[Arterial hypertension in glomerulonephritis].
A study is presented of the humoral mechanisms of arterial hypertension, clinical course and morphological changes of the renal tissue as evidenced by kidney biopsy data in 203 patients with mesangial proliferative glomerulonephritis (GN) depending on the presence of arterial hypertension and time of its development. The development of arterial hypertension is caused by genetic factors and is similar to hypertensive disease by its humoral indices. The mechanisms of initiation of arterial hypertension are also similar at the onset and final stage of GN. The morphological reflexion of arterial hypertension is sclerosis of the medullar layer of the kidneys. The hypertensive syndrome in GN should be considered from the viewpoint of its appearance as it determines the course of nephropathy.
B I
BI
Shulutko
D000328:Adult; D001707:Biopsy, Needle; D002908:Chronic Disease; D005260:Female; D015432:Glomerulonephritis, Membranoproliferative / Q000150:complications* / Q000473:pathology; D006801:Humans; D006973:Hypertension / Q000209:etiology* / Q000473:pathology; D007668:Kidney / Q000473:pathology; D008297:Male; D012598:Sclerosis / Q000473:pathology; D009626:Terminology as Topic
Arterial'naia gipertenziia pri glomerulonefrite.