The pathogenesis of slow virus infections: molecular analyses.
Slow infections raise some novel issues in understanding the pathogenesis of viral diseases that have been increasingly addressed at the molecular level with in situ hybridization. These developments are reviewed as the framework for discussion of the realized and potential impact of these investigations for major neurological afflictions of humans and for AIDS.
A T
AT
Haase
D000163:Acquired Immunodeficiency Syndrome / Q000382:microbiology; D000328:Adult; D000368:Aged; D000544:Alzheimer Disease / Q000382:microbiology; D000818:Animals; D004247:DNA / Q000032:analysis; D004279:DNA, Viral / Q000378:metabolism; D005786:Gene Expression Regulation; D006801:Humans; D008242:Lysogeny; D008459:Measles virus / Q000235:genetics*; D009693:Nucleic Acid Hybridization; D012367:RNA, Viral / Q000032:analysis; D012194:RNA-Directed DNA Polymerase / Q000378:metabolism; D012608:Scrapie / Q000235:genetics / Q000382:microbiology*; D012897:Slow Virus Diseases / Q000209:etiology*; D014790:Visna-maedi virus / Q000472:pathogenicity
D004279:DNA, Viral; D012367:RNA, Viral; D004247:DNA; D012194:RNA-Directed DNA Polymerase