title: Platelet derived growth factor (PDGF) and prostaglandins (PGE1, PGI2) as modulators of the atherogenetic process.
abstract: It is concluded that the interaction between platelets, the PDGF- and PG-formation may be of key-importance in human atherosclerosis, not only by regulation of smooth muscle cell proliferation and extracellular matrix production but also in cellular lipid metabolism.
authors: Atherosclerosis Research Group (ASF) Vienna, Austria.
authors: H
authors: H
authors: Sinzinger
authors: T
authors: T
authors: Zidek
authors: P
authors: P
authors: Fitscha
authors: J
authors: J
authors: Kaliman
authors: G
authors: G
authors: Steurer
mesh_terms: D000527:Alprostadil / Q000502:physiology*; D000818:Animals; D001161:Arteriosclerosis / Q000097:blood / Q000503:physiopathology*; D001792:Blood Platelets / Q000502:physiology*; D002455:Cell Division; D011464:Epoprostenol / Q000502:physiology*; D005109:Extracellular Matrix / Q000502:physiology; D066298:In Vitro Techniques; D050356:Lipid Metabolism; D009131:Muscle, Smooth, Vascular / Q000166:cytology / Q000502:physiology; D010982:Platelet-Derived Growth Factor / Q000502:physiology*; D011817:Rabbits
chemical_list: D010982:Platelet-Derived Growth Factor; D011464:Epoprostenol; D000527:Alprostadil