[Various structural and functional mechanisms of development of peptic ulcers after vagotomy].
Late restoration after subphrenic vagotomy of the acid- and enzyme-producing gastric functions, sharp decrease in cholinesterase activity of cholinergic nerve fibers, decrease of luminescence of the adrenergic nerve terminals with subsequent development in patients of hypersecretion and peptic anastomotic ulcer in association with normalization of the structure of the parietal and main cells are the result of gastric reinnervation.
Savinskiĭ
G A
GA
Khudaĭbergenov
R U
RU
Mansurov
B M
BM
D000328:Adult; D004381:Duodenal Ulcer / Q000473:pathology / Q000503:physiopathology / Q000601:surgery*; D005260:Female; D005744:Gastric Acid / Q000378:metabolism; D005753:Gastric Mucosa / Q000378:metabolism*; D006801:Humans; D008297:Male; D010295:Parietal Cells, Gastric / Q000473:pathology*; D010434:Pepsin A / Q000378:metabolism; D010437:Peptic Ulcer / Q000209:etiology*; D011183:Postoperative Complications / Q000209:etiology*; D012008:Recurrence; D015167:Vagotomy, Truncal / Q000009:adverse effects*
D010434:Pepsin A
Nekotorye strukturnye i funktsiona'lnye mekhanizmy voznikno- pepticheskikh iazv posle vagotomii.