Sodium retention in heart failure.
It is apparent therefore that a large variety of neurohumoral influences affect renal hemodynamics and the renal tubular reabsorption of sodium and water in response to cardiac failure and thereby contribute to the development of a "salt-avid" kidney, circulatory congestion, and edema formation. These influences are summarized schematically in Figure 1.
Cannon
P J
PJ
Department of Medicine, College of Physicians and Surgeons, Columbia University, New York, New York.
D001127:Arginine Vasopressin / Q000378:metabolism; D009320:Atrial Natriuretic Factor / Q000502:physiology; D004487:Edema / Q000378:metabolism; D006333:Heart Failure / Q000209:etiology / Q000378:metabolism* / Q000503:physiopathology; D006801:Humans; D007668:Kidney / Q000378:metabolism* / Q000503:physiopathology; D012084:Renin-Angiotensin System; D012964:Sodium / Q000097:blood / Q000378:metabolism*
D001127:Arginine Vasopressin; D009320:Atrial Natriuretic Factor; D012964:Sodium